Chapter 11 Extended Lecture Outline




Chapter Outline

INTRODUCTION

		All Organisms Grow and Reproduce

		All Species Pass Their Hereditary Information on to Their Offspring	fig 11.1

CELL DIVISION IN BACTERIA

		Binary Fission Is Bacterial Cell Division
			Genome replicated early in the life of the cell	fig 11.2
				Copying of the DNA circle occurs at the replication origin
				Requires a battery of enzymes
				End result:  two side-by-side circles of DNA on membrane
			Composition of the bacterial genome 
				Exists as one double-stranded circle of DNA
				Attached to one point on the interior of the cell membrane

		Division Initiated by Growth of the Cell to a Certain Size
			New plasma membrane and cell wall materials laid down
			Growing membrane pinches inward, cell constricted in two	fig 11.3
			Each cell contains a copy of the genome

CELL DIVISION IN EUKARYOTES

		Eukaryotic Genome Is Larger and More Complex than Bacterial Genome
			DNA located within linear chromosomes
			DNA forms a complex with histone proteins and is tightly coiled	fig 11.4

		The Structure of Eukaryotic Chromosomes
			Chromosomes first observed in dividing salamander larvae cells
			The number of chromosomes varies within species	tbl 11.1
			Chromosomes are composed of chromatin
				Complex of 40% DNA and 60% protein
				Contains some RNA since DNA is the site of RNA synthesis
			DNA exists as a long double-stranded fiber
			DNA coiled to fit into a smaller space than otherwise possible
				DNA resembles a string of beads	fig 11.5
				DNA is coiled around histone polypeptides every 200 nucleotides
				Eight histones form a core called a nucleosome
				Basic, positively charged histones attract negatively charged DNA
				String of nucleosomes further wrapped into supercoils
			Heterochromatin
				Highly condensed portions of chromatin
				Some portions permanently condensed to prevent DNA expression
			Euchromatin
				Remainder of chromatin condensed only during cell replication
				Movement of chromosomes facilitated by packaging
				DNA is uncondensed to allow for gene expression
			Chromosomes vary widely in appearance
				Position of the centromere
				Relative length of the arms on either side of the centromere
				Size and staining properties
				Position of additional constricted regions along arms	fig 11.6
			Karyotype:  array of an individual's chromosomes	fig 11.7
				In humans, blood sample collected, cells induced to divide
				Chemicals stop division at metaphase when chromosomes are most condensed
				Contents spread out, stained and then photographed
				Chromosomes cut out and arranged in order
			Karyotypes may reflect genetic abnormalities

		How Many Chromosomes Are in a Cell?
			All human body cells are diploid 
				Contain 46 chromosomes composed of 23 pairs
				Pairs are nearly identical and are called homologues
			Human gametes have haploid complement with 23 chromosomes
			Before division each of the two homologues replicates 	fig 11.8
				Produce two identical copies called sister chromatids
				Chromatids remain joined together at the centromere
				Cells have 46 replicated chromosomes each with two chromatids
					Possess 46 centromeres
					Four sets of genetic material:  23 pairs x 2 chromatids each
					Number of chromosomes indicated by number of centromeres

THE CELL CYCLE

		Cycle  of Cell Growth and Division Has Five Stages
			G1 phase:  primary growth phase
			S phase:  genome replica synthesized
			G2 phase:  preparations made for genomic separation
				Replication of mitochondria and other organelles
				Chromosome condensation
				Restructuring of microtubules and assembly at spindle
			M phase:  mitosis
				Microtubular apparatus assembled
				Sister chromatids move apart from one another
			C phase:  cytokinesis
				Physical division of the cell, creates two daughter cells
				Animal spindle helps position contracting cleavage furrow actin ring

		Duration of Cell Cycle Is Variable
			Embryos exhibit shortest cycles
				Divide as quickly as DNA can be replicated
				Half of cycle is S, half is M, virtually no G1 or G2
			Mature cells have longer cycles	fig 11.9
				Mammalian cell cycle averages 24 hours
				Growth occurs during G1 and G2
					G phases may be referred to as gap phases
					They separate the S phase from the M phase
				M phase takes only small portion of cycle
			Length of cycle variability is in G1
				Many cells pause in a G0 resting stage
				May remain there for days to years, some remain permanently
				Most body cells are in G0 at any one time
				Injury may stimulate some cells to enter G1 from G0

MITOSIS

		M and C Phases Are Readily Observed
			Constitute only small part of cell cycle	fig 11.10
			Mitosis subdivided into four continuous stages	fig 11.11
				Prophase
				Metaphase
				Anaphase
				Telophase

		Preparing for Mitosis:  Interphase
			G1 phase:  cells undergo major portion of growth
			S phase:  chromosome replicates to produce sister chromatids
				Remain attached at the centromere	fig 11.12
				Specific DNA sequence bound to a protein kinetochore	fig 11.13
				Location specific to each chromosome
			G2 phase:  chromosomes begin process of condensation
				Motor proteins involved in rapid, final condensation
				In G2 cells assemble machinery used to move chromosomes apart
					Animals replicate centriole, nuclear microtubule-organizing centers
					Eukaryotic cells synthesize tubulin, microtubule protein component

		Formation of the Mitotic Apparatus:  Prophase
			Individual condensed chromosomes become visible 
				Condensation continues through prophase
				Ribosomal RNA synthesis ceases, nucleolus disappears
			Microtubule apparatus made of spindle fibers begins to assemble
				In animal cells the two centrioles move apart
				Spindle apparatus, a bridge of microtubules, forms between them
				In plant cells, spindle apparatus forms without visible centrioles
				Position of spindle microtubules determines plane of cell division
				Division occurs at right angles through the spindle
			Nuclear envelope breaks down, materials absorbed by ER
			Animal cells form an arrangement called an aster	fig 11.14
				Centrioles at opposite poles extend radial array of microtubules
				Function to stiffen point of microtubular attachment
				Rigid plant cells do not form asters
			Second group of microtubules grow out from centromeres to poles
				Each chromosome possesses two kinetochores
				Two sets of microtubules extend from each chromosome
				Kinetochore of each sister chromatid connected to one pole
				Microtubules grow until they make contact with poles
				Sister chromatids won't separate if both connected to same pole

		Division of the Centromeres:  Metaphase
			Begins when pairs of sister chromatids align in center of the cell
			Chromosomes align along the metaphase plate	fig 11.15
				Not a physical structure
				Indicates where future axis of cell division occurs
			Centromeres are equidistant from each pole
			Centromeres divide at the end of metaphase
				Centromere splits in two
				All centromeres divide in synchrony 

		Separation of the Chromatids:  Anaphase
			Shortest phase, during which sister chromatids separate
			Chromatid drawn to pole to which it is attached
			Separation achieved by two simultaneous microtubular actions
				Poles move apart	fig 11.16
					Microtubular spindle fibers slide past one another
					Microtubules are anchored at poles which are pushed apart
					Chromatids attached to poles move apart as well
				Centromeres move toward poles
					Shortening process is not a contraction
					Microtubules shorten as tubulin subunits are removed
					Chromatids are therefore pulled toward poles

		Reformation of Nuclei:  Telophase
			Separation of chromatids completes partitioning of replicated genome
			Spindle apparatus is disassembled
			Tubulin units of microtubules are used to build new cytoskeleton
			Nuclear envelope re-forms around each new set of chromosomes
			Chromosomes begin to uncoil to allow gene expression
			rRNA genes begin transcription, nucleolus reappears

CYTOKINESIS

		Mitosis Complete at End of Telophase
			Replicated genome divided into two new nuclei at opposite ends of cell
			Cytoplasmic organelles assort to regions that will become separated
			Cleavage of the cell into two halves constitutes cytokinesis

		Cytokinesis in Plants and Animals Progresses Differently
			Animal cytokinesis 
				Cell is pinched in two by a constricting belt of microfilaments
					Actin filaments slide past one another
					Produces distinct cleavage furrow around circumference of cell	fig 11.17
				Furrow deepens until the cell is literally pinched in two	fig 11.18
			Plant cytokinesis
				Rigid cell wall, cannot be deformed by microfilament contraction
				Membrane components assembled in the cell interior	fig 11.19
					Occurs at right angles to the spindle apparatus
					Expanding partition called the cell plate
					Grows outward to the interior surface of the cell membrane
					Cellulose then added on the membrane making two new cells
				Middle lamellae:  space between cells impregnated with pectins
			In fungi and some protists mitosis is confined to the nucleus

CONTROL OF THE CELL CYCLE

		Events of Cell Cycle Coordinated Similarly in All Eukaryotes	fig 11.20
			Little change in processes over billions of years
			Human proteins can function when transferred to yeast cell

		General Strategy of Cell Cycle Control
			Goal of control is to optimize duration of cycle
			Internal clock control cannot provide sufficient flexibility
			Eukaryotes use a centralized controller based on cellular feedback
				Analogy:  furnace heating a house
				At points in cycle feedback determines if cycle continues or is delayed
			Three principle check points	fig 11.21
				Cell growth assessed at G1 check point
					Called START in yeasts
					If conditions favorable cell starts copying DNA, starting S phase
				DNA replication assessed at G2 check point
				Mitosis assessed at M check point

		Molecular Mechanisms of Cell Cycle Control
			Associated with interactions of proteins sensitive to cell conditions	fig 11.22
			Cyclin-dependent protein kinases (Cdk's)
				These enzymes phosphorylate serine and threonine of certain proteins
				Histones, nuclear membrane filaments, microtubule proteins at G2
			Cyclins 
				Bind to Cdk's, enable them to act as enzymes
				Are destroyed and resynthesized at each turn of cell cycle
			The G2 check point	fig 11.23
				Cell accumulates G2 cyclin (mitotic cyclin) during G2
					Binds to Cdk forming mitosis promoting factor, MPF
					MPF are phosphorylated and activated by cellular enzymes
					Positive feedback increases this activity, more MPF activated
					G2 ends when sufficient activated MPF
				Duration of M phase determined by MPF activity	fig 11.23
					MPF also activates proteins that destroy cyclin
					Degradation of G2 cyclin decreases activity of MPF, ending mitosis
			The G1 check point
				Similar to G2 control
				Yeasts compare volume of cytoplasm to size of genome
				In growth size increases, amount od DNA constant
				Threshold ration reached promoting cyclin production

		Controlling the Cell Cycle in Multicellular Eukaryotes
			Cells of multicellular organisms can't make individual decisions 
			Organization dependent limiting cell proliferation
			In cell culture cells stop dividing when sufficient numbers
				Growing cells take up growth factors like MPF	fig 11.24
				Example of positive regulatory signal
				If other cells take up factor, none left to trigger division in any cell

		Growth Factors and the Cell Cycle
			Growth factors trigger intracellular signalling systems
			Example:  fibroblasts
				Possess membrane receptors for platelet-derived growth factor, PDGF
				Binding PDGF and receptor initiates amplifying chain of events
				Tissue injury causes release of PDGF to promote healing
			Isolation of fifty growth factor proteins	tbl 11.2
				Each factor specifically recognized by specific cell surface receptor	fig 11.25
				Some affect broad range of cell types, PDGF and E (epidermal) GF
				Some affect only certain cell types, N (nerve) GF and erythropoietin
			Cells deprived of growth factors stop at G1, stay in G0

		Cancer and the Control of Cell Proliferation
			Proto-oncogenes normally stimulate cell division, positive approach
				Mutations causing them to overact change them into oncogenes
				Mutations are dominant
				Leads to excessive cell proliferation characteristic of cancer	fig 11.26
			30 different proto-oncogenes exist
			myc, fos, jun cause unrestrained cell growth and division
				myc in normal cell helps to regulate G1 check point	fig 11.27
				Genes also stimulate delayed response genes that produce cyclins, Cdk
			Tumor-suppressor genes normally inhibit cell division, negative approach
				Prevents binding of cyclins to Cdk, block passage through G1
				Mutations are recessive, unrestrained division if both copies mutated
			Retinoblastoma (Rb) gene is tumor-suppressor gene, causes rare eye cancer
				Normal gene is a cancer suppressor
				Rb gene encodes protein present in nucleus
				Rb protein is dephosphorylated in G0 phase	fig 11.24
					Binds regulatory proteins needed for cell proliferation
					Inhibit cell division
				When Rb is phosphorylated it releases regulatory proteins
					Cell division thus promoted
					Cells produce cyclins and Cdk, pass G1 check point

COMPARING CELL DIVISION IN EUKARYOTES AND PROKARYOTES

		Cell Division in Eukaryotes Is More Sophisticated than Division in Bacteria
			Chromosome movement is rapid and accurately partitions genome
			Bacterial replication depends on slow, uninterrupted membrane growth

		Different Processes Related to Size of Genome


[Return to Chapter 11 Page]
[Return to Chapter Tools Page]
[Return to Biology Home Page]

Search | How to Order | E-mail Us

Copyright ©1997 McGraw-Hill College Division